The majority of lateral ankle ligament injuries will resolve with non-operative care. Konradsen, et al. have showed that eighty percent of their patients with lateral ankle ligament injuries improved when treated with a course of supervised rehabilitation specifically aimed at proprioceptive and strength training with a seven year follow up . However, there are still about twenty percent of patients that will fail conservative management and go onto develop chronic instability. Both anatomic and non-anatomic techniques have been utilized to treat chronic lateral ankle instability in the literature.
The rationale for this particular modification to the original Broström technique is the improved surgical versatility of utilizing suture anchors for the repair of the ATFL, CFL, and the anterior capsule. First, the placement of the suture anchor at the footprints of the ATFL and CFL allows the surgeon to address each of the ligaments individually and the ability to repair them anatomically. Secondly, using anchors with sutures that are tied to the distal aspects of the ligaments will allow the surgeon the ability to better tighten the lateral ankle structures in comparison to primary repair. This particular technique also adds a third suture anchor approximately 1 cm above the ATFL insertion site, which is used to tighten the confluence of the proximal aspect of the ATFL ligament with the lateral ankle capsule. This is an important step because in patients with lateral ankle instability and tear of the ATFL, sometimes it is very difficult to distinguish the margin of the ATFL with the lateral capsule. Thus the addition of the third suture anchor allows further anatomic reinforcement of the repair without compromising ankle motion. Furthermore, the suture and the anchor used are absorbed over time, thus leaving no hardware in the ankle joint.
Our patient selection for this procedure is chronic lateral ankle instability refractory to at least a 6 months course of formal physical therapy. Both the talar tilt and anterior drawer stress radiographs are done in clinic with a mini fluoroscopy to document lateral instability. It is extremely important to compare the stress views with the contra-lateral ankle as some patients may have congenital laxity. Magnetic resonance imaging is also ordered on every patient to evaluate for intra-articular pathology and condition of the ATFL/CFL ligaments. Anyone with fractures, significant varus mal-alignment, severe osteoarthritis of the ankle, osteochondral dissecans lesions of the talus, and previous failed lateral ankle ligamenteous repair or reconstruction are not candidates for this procedure. Important points to remember during surgery include 1) meticulous elevation of the extensor retinaculum to leave a cuff of tissue for advancement, 2) careful and accurate periosteal dissection of the capsule off the fibula in order to preserve adequate length for repair, 3) always evaluate the intra-articular aspect of the ankle joint looking for loose bodies or OCD lesions, 4) when repairing the CFL, pay attention not to incorporate the peroneal tendon into the repair, and 5) if the CFL or ATFL is significantly shortened or not repairable, our bailout procedure of choice is an allograft reconstruction of both the ATFL and CFL. Furthermore, the most essential components to the success of this procedure is to tighten the three suture anchors as well as the extensor retinaculum with the patient's foot in neutral and slight eversion (Figure 7) which will further tighten up the lateral instability.
This technique was utilized by the authors in a series of high demand athletes with chronic lateral ankle instability and was able to return 94% of the patients to their previous sports activity level as demonstrated by the Tegner score. Also the average Karlsson ankle functional score was 92 +/- 5.2 and 95 +/- 3.1 at the one and two year post operative time frame, respectively. Only 3 patients out of 52 (6%) had a decrease in range of motion of greater than 5 degrees at the two year post operative follow up and there was no loss in subtalar motion. The major complication rate included a 6% re-rupture rate (3/53 patients) past the 1 year post operative period due to traumatic injuries in competition with no neurological injuries. We had several superficial wound infections that were treated and resolved with a course of oral antibiotics. Also a few patients had persistent ankle swelling, however, all of them resolved at the 6 months follow-up visit .