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Fig. 4 | Journal of Orthopaedic Surgery and Research

Fig. 4

From: The effect of extracorporeal shock wave on joint capsule fibrosis based on A2AR-Nrf2/HO-1 pathway in a rat extending knee immobilization model

Fig. 4

Protein expression levels of p-PKA/PKA, p-Nrf2/Nrf2, HO-1, A2AR and relative to β-Tubulin (The values are the mean ± standard deviation). A Western blotting bands of p-PKA/PKA, p-Nrf2/Nrf2, HO-1, A2AR and β-Tubulin proteins; B The quantitative analysis of p-PKA/PKA; C The quantitative analysis of p-Nrf2/Nrf2; D The quantitative analysis of HO-1; E The quantitative analysis of A2AR. a P < 0.05 versus the C group, b P < 0.05 versus the IM group, c P < 0.05 versus the NR group, d P < 0.05 versus the EI group. C Control group, IM Immobilization model group, NR Natural recovery group, EI Extracorporeal shock wave intervention group, CI Extracorporeal shock wave combined with A2AR antagonist SCH 58261 intervention. PKA Protein kinase A, HO-1 Haem oxidase-1, Nrf2 Neurotrophic factor e2-related factor 2, A2AR Adenosine A2A receptor, β-Tub β-Tubulin

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Journal of Orthopaedic Surgery and Research

ISSN: 1749-799X